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|EINECS:||216-140-2||Chemical Properties:||Crystalline Solid|
|Usage:||Ophthalmic Anticholinergic||Brand:||NJBN STEROID|
Tropicamide Active Pharmaceutical Ingredients 1508-75-4 Ophthalmic Anticholinergic
|solubility||45% (w/v) aq 2-hydroxypropyl-β-cyclodextrin: 4.3 mg/mL|
|Chemical Properties||Crystalline Solid|
|Usage||Indicated to induce mydriasis (dilation of the pupil) and cycloplegia (paralysis of the ciliary muscle of the eye) in diagnostic procedures, such as measurement of refractive errors and examination of the fundus of the eye.|
1.Tropicamide (Mydriacyl) is an anticholinergic used as a mydriatic.
Tropicamide is an antimuscarinic drug that produces short acting mydriasis (dilation of the pupil) and cycloplegia when applied as eye drops. It is used to allow better examination of the lens, vitreous humor, and retina. Due to its relatively short duration of effect (4–8 hours), it is typically used during eye examinations such as the dilated fundus examination, but it may also be used before or after eye surgery. Cycloplegic drops are often also used to treat anterior uveitis, decreasing risk of posterior synechiae and decreasing inflammation in the anterior chamber of the eye.
2.Right eye was instilled with tropicamide, leading to mydriasis and therefore anisocoria (unequal pupil size)
Anisocoria caused by Tropicamide instilled into the right eye only.
Tropicamide is occasionally administered in combination with p-hydroxyamphetamine (brand name Paremyd), which is a sympathomimetic. The use of the sympathomimetic drug causes the iris dilator muscle to be directly stimulated, causing increased dilation. In the United States, the sympathomimetic drop most commonly used along with tropicamide, is 2.5% phenylephrine hydrochloride (brand name AK-Dilate).
Tropicamide is a parasympatholytic agent, which acts by blocking the action of the parasympathetic nervous system. As acetylcholine is the neuro-humoral transmitter at the receptor site of the parasympathetic nervous system, tropicamide competes with acetylcholine for uptake at the receptor sites, thereby blocking its action. The results are mydriasis, due to unopposed action of the dilator pupillae, and cycloplegia.
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